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These data support the rationale for maternal or neonatal immunization, with acellular pertussis vaccines, to prevent life-threatening pertussis in early infancy. It was thought that administering pertussis vaccination to women in the last trimester [http://en.wikipedia.org/wiki/DDR1 DDR1] of pregnancy would interfere with the active immunity of the child. However, studies by Englund et?al. [23] did not find that maternal antibodies against PT interfered with the active immunization of the infant provided by the usual schedule of acellular pertussis vaccine. Consequently, even if interference occurs, it may not be relevant. It is still unknown whether maternal vaccination against pertussis is the best means of preventing pertussis in the first 6?months of life. However, Gall et?al. [24] have recently compared antibody levels against five proteins of B.?pertussis (PT, pertactin (PRN), FHA and FIM 2/3) in samples of umbilical cord blood taken from neonates born of mothers who had received Tdap vaccination during pregnancy or of mothers who were not vaccinated, and found [http://www.selleckchem.com/products/ABT-888.html check details] that the former had significantly higher concentrations of antibodies against PT (p?[http://www.selleckchem.com/products/Methazolastone.html Temozolomide molecular weight] is included in the vaccination schedule. The schedule for pneumococcal conjugate vaccine begins at the age of 2�C3?months, and complete protection against the pneumococcal serotypes included in the vaccine is not reached before the completion of the primary series (i.e. at the age of 5�C6?months). Furthermore, the 23-valent pneumococcal polysaccharide vaccine (PPV) is not recommended for healthy adults, and so pregnant women usually have poor pneumococcal antibody concentrations that are limited to the serotypes that have been the cause of a recent pneumococcal infection. To protect infants during the vulnerable first few months of life (i.e.