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[9, 11, 21] Hips with coxa magna have flattened head under the lateral part of the acetabulum with poor lateral coverage. The model for such hips yielded a small functional angle of load bearing with steeply falling stress in the medial direction (Fig. 2). The surface was constructed solely based on the curvature of the part that bears load. Other parts of the head do not lie on the articular surface, but this is immaterial in the model since they are not subject to stress. In Perthes, substantial head deformity may be present.[30] In our population the surface deviated from a sphere. In 3 Perthes hips out of 111 the deviation (��/r) was >20%, and in 6 it was between 10 and 20%. However, the effect on peak stress within both populations (test click here and control) was BML-190 higher in dysplastic hips.[11] Based on these results, it cannot be concluded which parameter is decisive for assessing the risk of early coxarthritis. Here, we report the difference in Gp but not peak stress of hips with Perthes disease compared to control hips. Multivariate analysis showed that the connection of the Herring lateral pillar Doxorubicin nmr score at the time of the disease and the follow-up Gp on the clinical status (HHS) of the hip was significant, but small, suggesting that after the appearance of symptoms, the disease progression is guided by several factors, with Gp (a biomechanical factor) and concomitant radiographic changes in the lateral pillar (morphological consequence of biomechanical factors) showing prominence. The significance of the impact of Gp and other biomechanical factors is marred by insufficiencies in the model, the limitations in imaging, and limited repeatability and accuracy of the method. Further, various radiographic classifications had poor inter-observer agreements/reliability.[31-33] Thus, the impact of the biomechanical factor in our analysis is underestimated. The control hips were contralateral to hips with Perthes disease. As Perthes may occur bilaterally[6, 34] our control group only approximates a group of healthy hips. The relevance of such an approximation is supported by the fact that hips matured after the onset (and treatment) of the disease and that Perthes disease or avascular necrosis of the head did not occur in contralateral hips. The contralateral hips' average radius and center-edge angle were comparable to those of normal hips.